{"@type":"Dataset","go_id":["https://identifiers.org/GO:0005739","https://identifiers.org/GO:0005737","https://identifiers.org/GO:0016310","https://identifiers.org/GO:0006915","https://identifiers.org/GO:0140354"],"go_kw":["mitochondrion","cytoplasm","phosphorylation","apoptosis","lipid uptake"],"integmet_study":"MTBLS3927","mesh_chemical_id":["https://identifiers.org/mesh:C033435","https://identifiers.org/mesh:D008055","https://identifiers.org/mesh:D006861","https://identifiers.org/mesh:D017382","https://identifiers.org/mesh:D005227","https://identifiers.org/mesh:D000255"],"mesh_chemical_pubtator_kw":["BAIBA","lipid","H2O2","ROS","H2 O 2","reactive oxygen species","fatty acid","beta-Aminoisobutyric Acid","ATP"],"mesh_disease_id":["https://identifiers.org/mesh:D006333","https://identifiers.org/mesh:D028361","https://identifiers.org/mesh:D020257","https://identifiers.org/mesh:D009203","https://identifiers.org/mesh:D064420"],"mesh_disease_pubtator_kw":["heart failure","HF","Mitochondrial Dysfunction","cardiac remodeling","myocardial infarction","adverse","mitochondrial dysfunction"],"mesh_gene_id":["https://identifiers.org/ncbigene:78975","https://identifiers.org/ncbigene:100526635"],"mesh_gene_pubtator_kw":["AMPK","miR-208b"],"ncbi_taxonomy_id":["https://identifiers.org/taxonomy:10116"],"ncbi_taxonomy_pubtator_kw":["rat","Rats","rats"],"source_id":"https://identifiers.org/metabolights:MTBLS3927","study_findings":"BAIBA reduces stress and apoptosis through miR-208b/AMPK pathway.","study_observation":"Cardiomyocyte apoptosis, energy metabolism, mitochondrial dysfunction, BAIBA effects.","study_summary":"BAIBA reduces cardiomyocyte stress via miR-208b/AMPK.","study_title_original":"Exercise-generated \u03b2-aminoisobutyric acid (BAIBA) reduces cardiomyocytes metabolic stress and apoptosis caused by mitochondrial dysfunction through the miR-208b/AMPK pathway"}