{"@type":"Dataset","integmet_study":"MTBLS3854","mesh_chemical_id":["https://identifiers.org/mesh:C010972","https://identifiers.org/mesh:D013654"],"mesh_chemical_pubtator_kw":["glycolaldehyde","Taurine"],"mesh_disease_id":["https://identifiers.org/mesh:D002869","https://identifiers.org/mesh:D011832","https://identifiers.org/mesh:D009369","https://identifiers.org/mesh:D002318"],"mesh_disease_pubtator_kw":["chromosomal aberration","radiation damage","cancer","cardiovascular diseases"],"ncbi_taxonomy_id":["https://identifiers.org/taxonomy:9606"],"ncbi_taxonomy_pubtator_kw":["human"],"source_id":"https://identifiers.org/metabolights:MTBLS3854","study_findings":"3-hydroxypropanoate and glycolaldehyde indicate radiation damage; taurine metabolism pathways differ under LLIR.","study_observation":"Metabolic phenotypic differences in LLIR and non-LLIR workers.","study_summary":"LLIR causes metabolic phenotype changes in workers.","study_title_original":"Long-term low-dose ionizing radiation induced chromosome-aberration-specific metabolic phenotype changes in radiation workers"}