{"@type":"Dataset","integmet_study":"MTBLS140","mesh_chemical_id":["https://identifiers.org/mesh:D005978","https://identifiers.org/mesh:C004656","https://identifiers.org/mesh:D007069"],"mesh_chemical_pubtator_kw":["glutathione","chloroacetaldehyde","ifosfamide"],"mesh_disease_id":["https://identifiers.org/mesh:D030321","https://identifiers.org/mesh:D064420","https://identifiers.org/mesh:D009369"],"mesh_disease_pubtator_kw":["Nephron Toxicity","toxicity","cancer"],"mesh_gene_id":["https://identifiers.org/ncbigene:468","https://identifiers.org/ncbigene:4780"],"mesh_gene_pubtator_kw":["ATF4","Nrf2"],"ncbi_taxonomy_id":["https://identifiers.org/taxonomy:9606"],"ncbi_taxonomy_pubtator_kw":["human"],"source_id":"https://identifiers.org/metabolights:MTBLS140","study_findings":"High concentration CAA treatment affects glutathione and oxidative stress response metabolites.","study_observation":"Chronic nephrotoxicity in RPTEC/TERT1 cells treated with chloroacetaldehyde.","study_summary":"Metabolome analysis for nephrotoxicity profiling using HPLC-ESI-MS.","study_title_original":"Metabolome analysis via an HPLC-ESI-MS-based experimental and computational pipeline for chronic nephron toxicity profiling"}