{"@type":"Dataset","go_id":["https://identifiers.org/GO:0065007","https://identifiers.org/GO:0006954","https://identifiers.org/GO:0006955","https://identifiers.org/GO:0023052"],"go_kw":["regulation","inflammation","immune response","signaling"],"integmet_study":"MTBLS1188","mesh_disease_id":["https://identifiers.org/mesh:D007239","https://identifiers.org/mesh:D007249","https://identifiers.org/mesh:D016778","https://identifiers.org/mesh:D008288"],"mesh_disease_pubtator_kw":["infection","inflammation","Falciparum malaria","malaria","inflammatory"],"ncbi_taxonomy_id":["https://identifiers.org/taxonomy:5833"],"ncbi_taxonomy_pubtator_kw":["Plasmodium falciparum"],"source_id":"https://identifiers.org/metabolights:MTBLS1188","study_findings":"Host variation affects inflammation and malaria progression; interferon signaling regulates host fate.","study_observation":"Parasite-host interactions and immune response in falciparum malaria.","study_summary":"Immune variation affects malaria outcomes.","study_title_original":"Immune variation leads to diverse outcomes in human malaria"}